medical:
diabetes deception
Our Deadly Diabetes DeceptionGreed and
dishonest science have promoted a lucrative worldwide
epidemic of diabetes that honesty and good science can
quickly reverse by naturally restoring the body's
blood-sugar control mechanism.
Extracted from Nexus Magazine, Volume 11, Number 4 (June-July
2004)
PO Box 30, Mapleton Qld 4560 Australia. editor@nexusmagazine.com
Telephone: +61 (0)7 5442 9280; Fax: +61 (0)7 5442 9381
From our web page at: www.nexusmagazine.com
by Thomas Smith © 2004
PO Box 7685
Loveland, CO 80537 USA
Email: Valley@healingmatters.com
Website: http://www.Healingmatters.com
(I regret, some of the earlier reference points are
missing.JB,Editor)
Introduction
If you are an American diabetic, your physician will
never tell you that most cases of diabetes are curable.
In fact, if you even mention the "cure" word
around him, he will likely become upset and irrational.
His medical school training only allows him to respond to
the word "treatment". For him, the
"cure" word does not exist. Diabetes, in its
modern epidemic form, is a curable disease and has been
for at least 40 years. In 2001, the most recent year for
which US figures are posted, 934,550 Americans died from
out-of-control symptoms of this disease.1
If you contract diabetes and depend upon orthodox medical
treatment, sooner or later you will experience one or
more of its symptoms as the disease rapidly worsens. It
is now common practice to refer to these symptoms as if
they were separable, independent diseases with separate,
unrelated treatments provided by competing medical
specialists.
It is true that many of these symptoms can and sometimes
do result from other causes; however, it is also true
that this fact has been used to disguise the causative
role of diabetes and to justify expensive, ineffective
treatments for these symptoms.
Epidemic Type II diabetes is curable. By the time you get
to the end of this article, you are going to know that.
You're going to know why it isn't routinely
being cured. And, you're going to know how to
cure it. You are also probably going to be angry at what
a handful of greedy people have surreptitiously done to
the entire orthodox medical community and to its trusting
patients.
The Diabetes Industry
Today's diabetes industry is a massive community that has
grown step by step from its dubious origins in the early
20th century. In the last 80 years it has become
enormously successful at shutting out competitive voices
that attempt to point out the fraud involved in modern
diabetes treatment. It has matured into a religion. And,
like all religions, it depends heavily upon the faith of
the believer.
The financial and political influence of this medical
community has almost totally subverted the original
intent of our regulatory agencies. They routinely approve
death-dealing, ineffective drugs with insufficient
testing.
The financial and political influence of this medical
community dominates our entire medical insurance
industry. This, in effect, neatly creates a special kind
of money that can only be spent within the orthodox
medical and drug industry. No other industry in the world
has been able to manage the politics of convincing people
to accept so large a part of their pay in a form that
often does not allow them to spend it as they see fit.
And then there are the various associations that solicit
annual donations to find a cure for their proprietary
disease. Every year they promise that a cure is just
around the cornerjust send more money! They
promoted the use of margarine as heart healthy, long
after it was well understood that margarine causes
diabetes and promotes heart failure.5 If
people ever wake up to the cure for diabetes that has
been suppressed for 40 years, these associations will
soon be out of business. But until then, they nonetheless
continue to need our support.
For 40 years, medical research has consistently shown
with increasing clarity that diabetes is a degenerative
disease directly caused by an engineered food supply that
is focused on profit instead of health. Although the
diligent can readily glean this information from a wealth
of medical research literature, it is generally otherwise
unavailable. Certainly this information has been, and
remains, largely unavailable in the medical schools that
train our retail doctors.
Prominent among the causative agents in our modern
diabetes epidemic are the engineered fats and oils that
are sold in today's supermarkets.
The first step to curing diabetes is to stop believing
the lie that the disease is incurable.
Diabetes History
In 1922, three Canadian Nobel Prize winners, Banting,
Best and Macleod, were successful in saving the life of a
fourteen-year-old diabetic girl in Toronto General
Hospital with injectable insulin.6 Eli
Lilly was licensed to manufacture this new wonder drug,
and the medical community basked in the glory of a job
well done.
It wasn't until 1933 that rumours about a new rogue form
of diabetes surfaced. This was in a paper presented by
Joslyn, Dublin and Marks and printed in the American
Journal of Medical Sciences. This paper,
"Studies on Diabetes Mellitus",7
discussed the emergence of a major epidemic of a disease
which looked very much like the diabetes of the early
1920s, only it did not respond to the wonder drug,
insulin. Even worse, sometimes insulin treatment killed
the patient.
This new disease became known as "insulin-resistant
diabetes" because it had the elevated blood sugar
symptom of diabetes but responded poorly to insulin
therapy. Many physicians had considerable success in
treating this disease through diet. A great deal was
learned about the relationship between diet and diabetes
in the 1930s and 1940s.
In 1950, the medical community became able to perform
serum insulin assays. These assays quickly revealed that
this new disease wasn't classic diabetes; it was
characterised by sufficient, often excessive, blood
insulin levels.
The problem was that the insulin was ineffective; it did
not reduce blood sugar. But since the disease had been
known as diabetes for almost 20 years, it was renamed
Type II diabetes. This was to distinguish it from the
earlier Type I diabetes, caused by insufficient insulin
production by the pancreas.
Had the dietary insights of the previous 20 years
dominated the medical scene from this point and into the
late 1960s, diabetes would have become widely recognised
as curable instead of merely treatable. Instead, in 1950,
a search was launched for another wonder drug to deal
with the Type II diabetes problem.
Cure versus Treatment
This new, ideal, wonder drug would be effective, like
insulin, in remitting obvious adverse symptoms of the
disease but not effective in curing the underlying
disease. Thus it would be needed continually for the
remaining life of the patient. It would have to be
patentable; that is, it could not be a natural medication
because these are non-patentable. Like insulin, it would
have to be highly profitable to manufacture and
distribute. Mandatory government approvals would be
required to stimulate physicians to prescribe it as a
prescription drug. Testing required for these approvals
would have to be enormously expensive to prevent other,
unapproved, medications from becoming competitive.
This is the origin of the classic medical protocol of
"treating the symptoms". By doing this, both
the drug company and the doctor could prosper in
business, and the patient, while not being cured of his
disease, was sometimes temporarily relieved of some of
his symptoms.
Additionally, natural medications that actually cured
disease would have to be suppressed. The more effective
they were, the more they would need to be suppressed and
their proponents jailed as quacks. After all, it wouldn't
do to have some cheap, effective, natural medication cure
disease in a capital-intensive monopoly market
specifically designed to treat symptoms without curing
disease.
Now it is clear why the "cure" word is so
vigorously suppressed by law. The FDA has extensive
Orwellian regulations that prohibit the use of the
"cure" word to describe any competing medicine
or natural substance. It is precisely because many
natural substances do actually both cure and prevent
disease that this word has become so frightening to the
drug and orthodox medical community.
The Commercial Value of
Symptoms
After the drug development policy was redesigned to focus
on ameliorating symptoms rather than curing disease, it
became necessary to reinvent the way drugs were marketed.
This was done in 1949 in the midst of a major epidemic of
insulin-resistant diabetes.
So, in 1949, the US medical community reclassified the
symptoms of diabetes10 along with many other disease symptoms into
diseases in their own right. With this reclassification
as the new basis for diagnosis, competing medical
speciality groups quickly seized upon related groups of
symptoms as their own proprietary symptoms set.
Thus the heart specialist, endocrinologist, allergist,
kidney specialist and many others started to treat the
symptoms for which they felt responsible. As the
underlying cause of the disease was widely ignored, all
focus on actually curing anything was completely lost.
Heart failure, for example, which had previously been
understood often to be but a symptom of diabetes, now
became a disease not directly connected to diabetes. It
became fashionable to think that diabetes "increased
cardiovascular risk". The causal role of a failed
blood-sugar control system in heart failure became
obscured.
Consistent with the new medical paradigm, none of the
treatments offered by the heart specialist actually
cures, or is even intended to cure, their proprietary
disease. For example, the three-year survival rate for
bypass surgery is almost exactly the same as if no
surgery was undertaken.11
Today, over half of the people in America suffer from one
or more symptoms of this disease. In its beginnings, it
became well known to physicians as Type II diabetes,
insulin-resistant diabetes, insulin resistance,
adult-onset diabetes or, more rarely, hyperinsulinaemia.
One third of the US population is morbidly obese; half of
the population is overweight. Type II diabetes, also
called adult-onset diabetes, now appears routinely in
six-year-old children.
Many degenerative diseases can be traced to a massive
failure of the endocrine system. This was well known to
the physicians of the 1930s as insulin-resistant
diabetes. This basic underlying disorder is known to be a
derangement of the blood-sugar control system by badly
engineered fats and oils. It is exacerbated and
complicated by the widespread lack of other essential
nutrition that the body needs to cope with the metabolic
consequences of these poisons.
All fats and oils are not equal. Some are healthy and
beneficial; many, commonly available in the supermarket,
are poisonous. The health distinction is not between
saturated and unsaturated, as the fats and oils industry
would have us believe. Many saturated oils and fats are
highly beneficial; many unsaturated oils are highly
poisonous. The important health distinction is between natural
and engineered.
There exists great dishonesty in advertising in the fats
and oils industry. It is aimed at creating a market for
cheap junk oils such as soy, cottonseed and rapeseed
oils.
With an informed and aware public, these oils would have
no market at all, and the USAindeed, the
worldwould have far fewer cases of diabetes.
Epidemiological Lifestyle Link
As early as 1901, efforts had been made to manufacture
and sell food products by the use of automated factory
machinery because of the immense profits that were
possible. Most of the early efforts failed because people
were inherently suspicious of food that wasn't farm fresh
and because the technology was poor.
Margarine was introduced and was bitterly opposed by the
dairy states in the USA. With the advent of the
Depression of the 1930s, margarine, Crisco and a host of
other refined and hydrogenated products began to make
significant penetration into the food markets of America.
Support for dairy opposition to margarine faded during
World War II because there wasn't enough butter for the
needs of both the civilian population and the military.13 At this
point, the dairy industry, having lost much support,
simply accepted a diluted market share and concentrated
on supplying the military.
Flax oils and fish oils, which were common in the stores
and considered dietary staples before the American
population became diseased, have disappeared from the
shelf. The last supplier of flax oil to the major
distribution chains was Archer Daniels Midland, and it
stopped producing and supplying the product in 1950.
More recently, one of the most important of the
remaining, genuinely beneficial, fats was subjected to a
massive media disinformation campaign that portrayed it
as a saturated fat that causes heart failure. As a
result, it has virtually disappeared from the supermarket
shelves. Thus was coconut oil removed from the food chain
and replaced with soy oil, cottonseed oil and rapeseed
oil.14 Our parents and grandparents would never have
swapped a fine, healthy oil like coconut oil for these
cheap, junk oils. It was shortly after this successful
media blitz that the US populace lost its war on fat. For
many years, coconut oil had been our most effective
dietary weight-control agent.
The history of the engineered adulteration of our
once-clean food supply exactly parallels the rise of the
epidemic of diabetes and hyperinsulinaemia now sweeping
the United States as well as much of the rest of the
world.
The second step to a cure for this disease epidemic is to
stop believing the lie that our food supply is safe and
nutritious.
The Nature of the Disease
Diabetes is classically diagnosed as a failure of the
body to metabolise carbohydrates properly. Its defining
symptom is a high blood-glucose level. Type I diabetes
results from insufficient insulin production by the
pancreas. Type II diabetes results from ineffective
insulin. In both types, the blood-glucose level remains
elevated. Neither insufficient insulin nor ineffective
insulin can limit post-prandial (after-eating) blood
sugar to the normal range. In established cases of Type
II diabetes, these elevated blood sugar levels are often
preceded and accompanied by chronically elevated insulin
levels and by serious distortions of other endocrine
hormonal markers.
The ineffective insulin is no different from effective
insulin. Its ineffectiveness lies in the failure of
the cell population to respond to it. It is not the
result of any biochemical defect in the insulin itself.
Therefore, it is appropriate to note that this is a
disease that affects almost every cell in the 70 trillion
or so cells of the body. All of these cells are
dependent upon the food that we eat for the raw materials
they need for self repair and maintenance.
The classification of diabetes as a failure to metabolise
carbohydrates is a traditional classification that
originated in the early 19th century when little was
known about metabolic diseases or processes.15 Today,
with our increased knowledge of these processes, it would
appear quite appropriate to define Type II diabetes more
fundamentally as a failure of the body to metabolise fats
and oils properly. This failure results in a loss of
effectiveness of insulin and in the consequent failure to
metabolise carbohydrates. Unfortunately, much medical
insight into this matter, except at the research level,
remains hampered by its 19th-century legacy.
Thus Type II diabetes and its early hyperinsulinaemic
symptoms are whole-body symptoms of this basic cellular
failure to metabolise glucose properly. Each cell of the
body, for reasons which are becoming clearer, finds
itself unable to transport glucose from the bloodstream
to its interior. The glucose then remains in the
bloodstream, or is stored as body fat or as glycogen, or
is otherwise disposed of in urine.
ORGANIC PROCESS:
It appears that when insulin binds to a cell
membrane receptor, it initiates a complex cascade
of biochemical reactions inside the cell. This
causes a class of glucose transporters known as
GLUT4 molecules to leave their parking area
inside the cell and travel to the inside surface
of the plasma cell membrane.
When in the membrane, they migrate to special
areas of the membrane called caveolae areas.16
There, by another series of biochemical
reactions, they identify and hook up with glucose
molecules and transport them into the interior of
the cell by a process called endocytosis. Within
the cell's interior, this glucose is then burned
as fuel by the mitochondria to produce energy to
power cellular activity. Thus these GLUT4
transporters lower glucose in the bloodstream by
transporting it out of the bloodstream into all
the cells of the body.
Many of the molecules involved in these glucose-
and insulin-mediated pathways are lipids; that
is, they are fatty acids. A healthy plasma cell
membrane, now known to be an active player in the
glucose scenario, contains a complement of
cis-type w=3 unsaturated fatty acids.17
This makes the membrane relatively fluid and
slippery. When these cis- fatty acids are
chronically unavailable because of our diet,
trans- fatty acids and short- and medium-chain
saturated fatty acids are substituted in the cell
membrane. These substitutions make the cellular
membrane stiffer and more sticky, and inhibit the
glucose transport mechanism.18
Thus, in the absence of sufficient cis omega 3
fatty acids in our diet, these fatty acid
substitutions take place, the mobility of the
GLUT4 transporters is diminished, the interior
biochemistry of the cell is changed and glucose
remains elevated in the bloodstream.
Elsewhere in the body, the pancreas secretes
excess insulin, the liver manufactures fat from
the excess sugar, the adipose cells store excess
fat, the body goes into a high urinary mode,
insufficient cellular energy is available for
bodily activity and the entire endocrine system
becomes distorted. Eventually, pancreatic failure
occurs, body weight plummets and a diabetic
crisis is precipitated.
Although there remains much work to be done to
elucidate fully all of the steps in all of these
pathways, this clearly marks the beginning of a
biochemical explanation for the known
epidemiological relationship between cheap,
engineered dietary fats and oils and the onset of
Type II diabetes. |
Orthodox Medical Treatment
After the diagnosis of diabetes, modern orthodox medical
treatment consists of either oral hypoglycaemic agents or
insulin.
Oral
hypoglycaemic agents
In 1955, oral hypoglycaemic drugs were
introduced. Currently available oral
hypoglycaemic agents fall into five
classifications according to their biophysical
mode of action.19 These classes
are: biguanides; glucosidase inhibitors;
meglitinides; sulphonylureas; and
thiazolidinediones.
The biguanides lower
blood sugar in three ways. They inhibit the
normal release by the liver of its glucose
stores, they interfere with intestinal absorption
of glucose from ingested carbohydrates, and they
are said to increase peripheral uptake of
glucose.
The glucosidase inhibitors
are designed to inhibit the amylase enzymes
produced by the pancreas and which are essential
to the digestion of carbohydrates. The theory is
that if the digestion of carbohydrates is
inhibited, the blood sugar level cannot be
elevated.
The meglitinides are
designed to stimulate the pancreas to produce
insulin in a patient that likely already has an
elevated level of insulin in their bloodstream.
Only rarely does the doctor even measure the
insulin level. Indeed, these drugs are frequently
prescribed without any knowledge of the
pre-existing insulin level. The fact that an
elevated insulin level is almost as damaging as
an elevated glucose level is widely ignored.
The sulphonylureas are
another pancreatic stimulant class designed to
stimulate the production of insulin. Serum
insulin determinations are rarely made by the
doctor before he prescribes these drugs. They are
often prescribed for Type II diabetics, many of
whom already have elevated ineffective insulin.
These drugs are notorious for causing
hypoglycaemia as a side effect.
The thiazolidinediones
are famous for causing liver cancer. One of them,
Rezulin, was approved in the USA through devious
political infighting, but failed to get approval
in the UK because it was known to cause liver
cancer. The doctor who had responsibility to
approve it at the FDA refused to do so. It was
only after he was replaced by a more compliant
official that Rezulin gained approval by the FDA.
It went on to kill well over 100 diabetes
patients and cripple many others before the fight
to get it off the market was finally won. Rezulin
was designed to stimulate the uptake of glucose
from the bloodstream by the peripheral cells and
to inhibit the normal secretion of glucose by the
liver. The politics of why this drug ever came
onto market, and then remained in the market for
such an unexplainable length of time with
regulatory agency approval, is not clear.20
As of April 2000, lawsuits
commenced to clarify this situation.21 |
.
Insulin
Today, insulin is prescribed for both the Type I and Type
II diabetics. Injectable insulin substitutes for the
insulin that the body no longer produces. Of course, this
treatment, while necessary for preserving the life of the
Type I diabetic, is highly questionable when applied to
the Type II diabetic.
It is important to note that neither insulin nor any
of these oral hypoglycaemic agents exerts any curative
action whatsoever on any type of diabetes. None of these
medical strategies is designed to normalise the cellular
uptake of glucose by the cells that need it to power
their activity.
The prognosis with this orthodox treatment is increasing
disability and early death from heart or kidney failure
or the failure of some other vital organ.
Alternative Medical Treatment
The third step to a cure for this disease is to become
informed and to apply an alternative methodology that is
soundly based upon good science.
Effective alternative treatment that directly leads to a
cure is available today for some Type I and for many Type
II diabetics. About 5% of the diabetic population suffers
from Type I diabetes; about 95% has Type II diabetes.22
Gestational diabetes is simply ordinary diabetes
contracted by a woman who is pregnant.
For the Type I diabetic, an alternative methodology for
the treatment of Type I diabetes is now available. It was
developed in modern hospitals in Madras, India, and
subjected to rigorous double-blind studies to prove its
efficacy.23 It operates to restore normal pancreatic beta
cell function so that the pancreas can again produce
insulin as it should. This approach apparently was
capable of curing Type I diabetes in over 60% of the
patients on whom it was tested. The major complication
lies in whether the antigens that originally led to the
autoimmune destruction of these beta cells have
disappeared from or remain in the body. If they remain, a
cure is less likely; if they have disappeared, the cure
is more likely. For reasons already discussed, this
methodology is not likely to appear in the United States
any time soon, and certainly not in the American orthodox
medical community.
The goal of any effective alternative program is to
repair and restore the body's own blood-sugar control
mechanism. It is the malfunctioning of this mechanism
that, over time, directly causes all of the many
debilitating symptoms that make orthodox treatment so
financially rewarding for the diabetes industry. For Type
II diabetes, the steps in the program are:24
Repair the faulty blood sugar control
system. This is done simply by substituting
clean, healthy, beneficial fats and oils in the diet for
the pristine-looking but toxic trans-isomer mix
found in attractive plastic containers on supermarket
shelves. Consume only flax oil, fish oil and occasionally
cod liver oil until blood sugar starts to stabilise. Then
add back healthy oils such as butter, coconut oil, olive
oil and clean animal fat. Read labels; refuse to consume
cheap junk oils when they appear in processed food or on
restaurant menus. Diabetics are chronically short of
minerals; they need to add a good-quality, broad-spectrum
mineral supplement to the diet.
Control blood sugar
manually during the recovery cycle. Under
medical supervision, gradually discontinue all oral
hypoglycaemic agents along with any additional drugs
given to counteract their side effects. Develop natural
blood-sugar control by the use of glycaemic tables, by
consuming frequent small meals (including fibre-rich
foods), by regular post-prandial exercise, and by the
complete avoidance of all sugars along with the judicious
use of only non-toxic sweeteners.25 Avoid
alcohol until blood sugar stabilises in the normal range.
Keep score by using a pinprick-type glucose meter. Keep
track of everything you do with a medical diary.
Restore a proper balance
of healthy fats and oils when the blood sugar controller
again works. Permanently remove from the diet
all cheap, toxic, junk fats and oils as well as the
processed and restaurant foods that contain them. When
the blood sugar controller again starts to work
correctly, gradually introduce additional healthy foods
to the diet. Test the effect of these added foods by
monitoring blood sugar levels with the pinprick-type
blood sugar monitor. Be sure to include the results of
these tests in your diary also.
Continue the program
until normal insulin values are also restored
after blood sugar levels begin to stabilise in the normal
region. Once blood sugar levels fall into the normal
range, the pancreas will gradually stop overproducing
insulin. This process will typically take a little longer
and can be tested by having your physician send a sample
of your blood to a lab for a serum insulin determination.
A good idea is to wait a couple of months after blood
sugar control is restored and then have your physician
check your insulin level. It's nice to have blood sugar
in the normal range; it's even nicer to have this
accomplished without excess insulin in the bloodstream.
Separately repair the
collateral damage done by the disease. Vascular
problems caused by a chronically elevated glucose level
will normally reverse themselves without conscious
effort. The effects of retinopathy and of peripheral
neuropathy, for example, will usually self repair.
However, when the fine capillaries in the basement
membranes of the kidneys begin to leak due to chronic
high blood glucose, the kidneys compensate by laying down
scar tissue to prevent the leakage. This scar tissue
remains even after the diabetes is cured, and is the
reason why the kidney damage is not believed to self
repair.
A word of warning
When retinopathy develops, there may be a temptation to
have the damage repaired by laser surgery. This laser
technique stops the retinal bleeding by creating scar
tissue where the leaks have developed. This scar tissue
will prevent normal healing of the fine capillaries in
the eye when the diabetes is reversed. By reversing the
diabetes instead of opting for laser surgery, there is an
excellent chance that the eye will heal completely.
However, if laser surgery is done, this healing will
always be complicated by the scar tissue left by the
laser.
The arterial and vascular damage done by years of
elevated sugar and insulin and by the proliferation of
systemic candida will slowly reverse due to improved
diet. However, it takes many years to clean out the
arteries by this form of oral chelation. Arterial damage
can be reversed much more quickly by using intravenous
chelation therapy.26 What would normally take many years through
diet alone can often be done in six months with
intravenous therapy. This is reputed to be effective over
80% of the time. For obvious reasons, don't expect your
doctor to approve of this, particularly if he's a heart
specialist.
Recovery Time
The prognosis is usually swift recovery from the disease
and restoration of normal health and energy levels in a
few months to a year or more. The length of time that it
takes to effect a cure depends upon how long the disease
was allowed to develop.
For those who work quickly to reverse the disease after
early discovery, the time is usually a few months or
less. For those who have had the disease for many years,
this recovery time may lengthen to a year or more. Thus,
there is good reason to get busy reversing this disease
as soon as it becomes clearly identified.
By the time you get to this point in this article, and if
we've done a good job of explaining our diabetes
epidemic, you should know what causes it, what orthodox
medical treatment is all about, and why diabetes has
become a national and international disgrace.
Of even greater importance, you have become acquainted
with a self-help program that has demonstrated great
potential to actually cure this disease.
About the Author:
Thomas Smith is a reluctant medical investigator, having
been forced into curing his own diabetes because it was
obvious that his doctor would not or could not cure it.
He has published the results of his successful diabetes
investigation in his self-help manual, Insulin:
Our Silent Killer, written for the
layperson but also widely valued by the medical
practitioner. This manual details the steps required to
reverse Type II diabetes and references the work being
done with Type I diabetes. The book may be purchased from
the author at PO Box 7685, Loveland, Colorado 80537, USA
(North American residents send $US25.00; overseas
residents should contact the author for payment and
shipping instructions).
Thomas Smith has also posted a great deal of useful
information about diabetes on his website, http://www.Healingmatters.
com. He can be contacted by
telephone at +1 (970) 669 9176 and by email at valley@healingmatters.com.
Endnotes:
1. National Center for Health Statistics, "Fast
Stats", Deaths/Mortality Preliminary 2001 data
2. Dr Herbert Ley, in response to a question from Senator
Edward Long about the FDA during US Senate hearings in
1965
3. Eisenberg, David M., MD, "Credentialing
complementary and alternative medical providers", Annals
of Internal Medicine 137(12):968 (December 17, 2002)
4. American Diabetes Association and the American
Dietetic Association, The Official Pocket Guide to
Diabetic Exchanges, McGraw-Hill/Contemporary
Distributed Products, newly updated March 1, 1998
5. American Heart Association, "How Do I Follow a
Healthy Diet?", American Heart Association
National Center (7272 Greenville Avenue, Dallas, Texas
75231-4596, USA), http://www.americanheart.org
6. Brown., J.A.C., Pears Medical Encyclopedia
Illustrated, 1971, p. 250
7. Joslyn, E.P., Dublin, L.I., Marks, H.H., "Studies
on Diabetes Mellitus", American Journal of
Medical Sciences 186:753-773 (1933)
8. "Diabetes Mellitus", Encyclopedia
Americana, Library Edition, vol. 9, 1966, pp. 54-56
9. American Heart Association, "Stroke (Brain
Attack)", August 28, 1998,
http://www.amhrt.org/ScientificHStats98/05stroke.html;
American Heart Association, "Cardiovascular Disease
Statistics", August 28, 1998,
http://www.amhrt.org/Heart_and_Stroke_A_Z_Guide/cvds.html;
"Statistics related to overweight and obesity",
http://niddk.nih.gov/health/nutrit/pubs/statobes.htm;
http://www.winltdusa.com/about/infocenter/
healthnews/articles/obesestats.htm
10. "Diabetes Mellitus", Encyclopedia
Americana, ibid., pp. 54-55
11. The Veterans Administration Coronary Artery Bypass
Co-operative Study Group, "Eleven-year survival in
the Veterans Administration randomized trial of coronary
bypass surgery for stable angina", New Eng. J.
Med. 311:1333-1339 (1984); Coronary Artery Surgery
Study (CASS), "A randomized trial of coronary artery
bypass surgery: quality of life in patients randomly
assigned to treatment groups", Circulation
68(5):951-960 (1983)
12. Trager, J., The Food Chronology, Henry Holt
& Company, New York, 1995 (items listed by date)
13. "Margarine", Encyclopedia Americana,
Library Edition, vol. 9, 1966, pp. 279-280
14. Fallon, S., Connolly, P., Enig, M.C., Nourishing
Traditions, Promotion Publishing, 1995;
Enig, M.C., "Coconut: In Support of Good Health
in the 21st Century", http://www.livecoconutoil.com/maryenig.htm
15. Houssay, Bernardo, A., MD, et al., Human
Physiology, McGraw-Hill Book Company, 1955, pp.
400-421
16. Gustavson, J., et al., "Insulin-stimulated
glucose uptake involves the transition of glucose
transporters to a caveolae-rich fraction within the
plasma cell membrane: implications for type II
diabetes", Mol. Med. 2(3):367-372 (May
1996)
17. Ganong, William F., MD, Review of Medical
Physiology, 19th edition, 1999, p. 9, pp. 26-33
18. Pan, D.A. et al., "Skeletal muscle membrane
lipid composition is related to adiposity and insulin
action", J. Clin. Invest. 96(6):2802-2808
(December 1995)
19. Physicians' Desk Reference, 53rd edition,
1999
20. Smith, Thomas, Insulin: Our Silent Killer, Thomas
Smith, Loveland, Colorado, revised 2nd
edition, July 2000, p. 20
21. Law Offices of Charles H. Johnson & Associates
(telephone 1 800 535 5727, toll free in North America)
22. American Heart Association, "Diabetes Mellitus
Statistics", http://www.amhrt.org
23. Shanmugasundaram, E.R.B. et al. (Dr Ambedkar
Institute of Diabetes, Kilpauk Medical College Hospital,
Madras, India), "Possible regeneration of the Islets
of Langerhans in Streptozotocin-diabetic rats given
Gymnema sylvestre leaf extract", J.
Ethnopharmacology 30:265-279 (1990);
Shanmugasundaram, E.R.B. et al., "Use of Gemnema
sylvestre leaf extract in the control of blood glucose in
insulin-dependent diabetes mellitus", J.
Ethnopharmacology 30:281-294 (1990)
24. Smith, ibid., pp. 97-123
25. Many popular artificial sweeteners on sale in the
supermarket are extremely poisonous and dangerous to the
diabetic; indeed, many of them are worse than the sugar
the diabetic is trying to avoid; see, for example, Smith,
ibid., pp. 53-58.
26. Walker, Morton, MD, and Shah, Hitendra, MD, Chelation
Therapy, Keats Publishing, Inc., New Canaan,
Connecticut, 1997, ISBN 0-87983-730-6
NEXUS
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